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. Author manuscript; available in PMC: 2009 Sep 14.

Published in final edited form as: Neurobiol Dis. 2007 Oct 16;29(2):336–353. doi: 10.1016/j.nbd.2007.09.009

Fig. 3 — β-amyloid pathology is reduced in Aβ_1–42-immunized PSAPP mice heterozygous for CD40. Mouse coronal brain sections were embedded in paraffin and stained with monoclonal human Aβ antibody (A), or were stained with congo red (B), and the hippocampus is shown. (C) Percentages [plaque area/total area; mean ± SD with n = 16 mice (8♂/8♀)] of Aβ antibody-immunoreactive Aβ plaques (top panel) and congo red-positive Aβ deposits (bottom panel) were calculated by quantitative image analysis for each brain region (CC/H: cingulate cortex and hippocampus; EC: entorhinal cortex) as indicated.

Fig. 3 — β-amyloid pathology is reduced in Aβ_1–42-immunized PSAPP mice heterozygous for CD40. Mouse coronal brain sections were embedded in paraffin and stained with monoclonal human Aβ antibody (A), or were stained with congo red (B), and the hippocampus is shown. (C) Percentages [plaque area/total area; mean ± SD with n = 16 mice (8♂/8♀)] of Aβ antibody-immunoreactive Aβ plaques (top panel) and congo red-positive Aβ deposits (bottom panel) were calculated by quantitative image analysis for each brain region (CC/H: cingulate cortex and hippocampus; EC: entorhinal cortex) as indicated.