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. 2009 Sep 7;3:184–190. doi: 10.2174/1874210600903010184

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© Moreira et al.; Licensee Bentham Open.

This is an open access article licensed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.

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Fig. (1) — Analysis of combined genotypes concerning TNFA (-308) and IL10 (-1082) polymorphisms in the different study groups. Individuals with the A+ genotype of the TNFA (-308) polymorphism are classified as “high TNF- alpha producers” and individuals with the A- genotype are classified as “low TNF- alpha producers”. Individuals with the G+ genotype of the IL10 (-1082) polymorphism are classified as “high IL-10 producers” and individuals with G- genotype are classified as “low IL-10 producers”. The “anti-inflammatory genotype” contains individuals classified as “low TNF- alpha and high IL-10” producers (A-/G+ for TNF- alpha and IL-10, respectively) and the “inflammatory genotype” contains individuals classified as “high TNF-alpha and low IL-10” producers (A+/G- for TNF- α and IL-10, respectively). Black bars (positive) represent individuals with the “inflammatory” or “anti-inflammatory” genotypes in respective graphs. Clear bars (negative) contain individuals that do not bear the specific combinations defined as “inflammatory” or “anti-inflammatory” genotypes. The clear bars in “inflammatory” graphs contains individuals with the genotypes (A+/G+ and A-/G- for TNF-alpha/IL-10) and in the “anti-inflammatory” graph, individuals that express A+/G+ and A+/G- for TNF-alpha/IL-10. No statistical difference was observed among groups, considering or not the habit of smoking (χ2 test, 3 x 2 contingency tables). C= Control, AP= aggressive periodontitis, CP= chronic periodontitis.