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. 2009 Sep;183(1):149–160. doi: 10.1534/genetics.109.103846

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Copyright © 2009 by the Genetics Society of America

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Figure 7.— — A model depicting a critical role for Esa1 and Rpd3L in coordinating the dynamic acetylation of H4K12. (A) Esa1 and Rpd3L control H4K12Ac for general transcriptional targets contributing to cell viability and growth. (B) Esa1 and Rpd3L contribute to rDNA and telomeric silencing. This relationship is not mediated specifically through H4K12 acetylation, but likely through a number of other targets. Sir2 deacetylation of H4K16 appears downstream of the role for Esa1 and Rpd3L. (C) Esa1 and Rpd3S, but not Rpd3L, may control acetylation at sites of DNA damage.