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. 2009 Jul 31;191(19):5930–5940. doi: 10.1128/JB.00703-09

FIG. 2.

FIG. 2.

Glucose catabolite repression-defective phenotypes of the S. griseus ΔcebR mutant. (A) β-Galactosidase activity on IS-GLX agar medium. (B) Halo formation by peptidoglycan degradation activity on YMP medium containing 1% glucose. (C to H) Morphology on YMP agar medium supplemented with no carbohydrates (C), 2.0% glucose (D), 1.5% glucose (E), 1.5% lactose (F), 1.0% glucose plus 0.5% lactose (G), and 1.0% glucose plus 0.5% lactose and 100 mM TES (pH 7.5) (H). 1, the wild-type strain harboring pIJ922; 2, the ΔcebR mutant harboring pIJ922; 3, the ΔcebR mutant harboring pIJ922-cebRwt; and 4, the ΔcebR mutant harboring pIJ922-cebRGRD1.