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. Author manuscript; available in PMC: 2009 Sep 28.

Published in final edited form as: J Mol Cell Cardiol. 2007 Feb 8;42(4):792–803. doi: 10.1016/j.yjmcc.2007.02.001

Figure-5 — Assessment of survival and angiomyogenesis in the infarcted myocardium. (A-B) Extensive survival of PKH-67 labeled SkM was observed in group-3 in the infarcted myocardium. (C-E) Immunostaining of rat heart tissue for fast skeletal myosin heavy chain expression showed extensive myogenesis in the infarct and peri-infarct areas in both (D) group-2 and (E) group-3 using (C) group-1 as control. Blood vessel density was assessed in the infarct (F-H) and peri-infarct areas (I-K) in all the groups which showed that (L) blood vessel density was significantly higher in group-3 as compared with groups-1 and -2.

Figure-5 — Assessment of survival and angiomyogenesis in the infarcted myocardium. (A-B) Extensive survival of PKH-67 labeled SkM was observed in group-3 in the infarcted myocardium. (C-E) Immunostaining of rat heart tissue for fast skeletal myosin heavy chain expression showed extensive myogenesis in the infarct and peri-infarct areas in both (D) group-2 and (E) group-3 using (C) group-1 as control. Blood vessel density was assessed in the infarct (F-H) and peri-infarct areas (I-K) in all the groups which showed that (L) blood vessel density was significantly higher in group-3 as compared with groups-1 and -2.