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. Author manuscript; available in PMC: 2009 Oct 5.
Published in final edited form as: Kidney Int. 1999 Jun;55(6):2390–2396. doi: 10.1046/j.1523-1755.1999.00466.x

Fig. 1. The percentage change in mean arterial blood pressure (BP), mean glomerular blood pressure (PGC), afferent and efferent arteriolar resistances (RA and RE, respectively), and the glomerular capillary ultrafiltration coefficient (Kf) in the normal anesthetized rat.

Fig. 1

Rats received acute systemic NO synthesis inhibition (NOSI) alone, with NMA (group I), combined endothelin blockade of ETA and ETB receptors and NOSI with bosentan and NMA (group II), combined NOSI and angiotensin II AT1 receptor blockade with losartan and NMA (group III), and NOSI during combined ETA, ETB, and AT1 receptor blockade (group IV). The asterisk denotes a significant difference in the response versus NOSI alone.