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. Author manuscript; available in PMC: 2010 Feb 3.
Published in final edited form as: Circulation. 2009 Jan 19;119(4):547–557. doi: 10.1161/CIRCULATIONAHA.108.788653

Figure 2.

Figure 2

Representative immunoblots (top) and densitometric quantification (bottom) demonstrating renal protein expression of phosphorylated Akt, phosphorylated eNOS, VEGF, HIF-1α, and angiopoietin-1 in normal, kidneys with renal artery stenosis (RAS), and RAS treated with autologous endothelial progenitor cells (RAS+EPC). Replenishment of EPC in RAS augmented the expression of vascular growth factors in the stenotic kidney, suggesting a pro-angiogenic stimulus. *p<0.05 vs. Normal. †p<0.05 vs. RAS, # p=0.079 vs. Normal.