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. 2009 Oct 6;106(42):17992–17997. doi: 10.1073/pnas.0909607106

Fig. 3.

Fig. 3.

GA treatment increases injury-induced rise of OPC numbers. (A) The number of PDGRα-positive OPCs were counted in the dorsal column of 10 sections, each spaced 100 μm apart and centered around the lesion epicenter. The sum per mouse was then plotted in A, where each value is from a separate mouse. There were 7 uninjured mice, 8 lysolecithin-vehicle mice, and 11 lysolecithin-GA animals. **, P < 0.01; ***, P < 0.001 (one way ANOVA with Bonferroni multiple comparison test). (B) Data of OPC numbers (mean ± SEM) in discrete sections around the lesion epicenter are displayed, to indicate that the increase in OPCs induced by GA is widespread across several sections containing demyelination. *, P < 0.05, one way ANOVA with Bonferroni multiple comparison test. All analyses were blinded.