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. 2009 Feb 6;2:9–16.

Table 2.

Pathophysiological processes that have been linked to the etiology acute lung injury post smoke inhalation

Airways oxidative stress Beta 2 – agonists
Airways cast formation Nitric oxide
Loss of pulmonary surfactants Eicosanoids – Prostaglandins
Airways bronchoconstriction – Thromboxane
Neutrophil activation/chemotaxis – Leukotrienes