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. 2009 Oct 28;4(10):e7650. doi: 10.1371/journal.pone.0007650

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Samuel, Latinkić.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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(A) animal cap injected with 0.5 ng of ΔTcf3 or 1 ng of Dkk-1, or uninjected control animal cap, were conjugated with anterior endoderm and analyzed for MHCα expression. The effect of Dkk-1 was quantified and shown below the image. In two additional experiments, Dkk-1 stimulated cardiogenesis 5- and 9-fold. (B) Dkk-1 cannot act in Nodal-independent manner, as it is blocked by CerS. The effectiveness of ΔTcf3 was shown by posteriorization of embryos (data not shown).