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1 treatment caused dose-dependent dephosphorylation of VEGFR-3 and decreased phosphorylation of FAK. A and B, Western blot analysis of MCF7-VEGFR-3 breast cancer cells and BT474 breast cancer cells after 24 h of treatment with increasing doses of 1. VEGFR-3 phosphorylation was analyzed. C and D, Immunoprecipitation of FAK and Western blot analysis with anti-phosphotyrosine antibody 4G10 of MCF7-VEGFR-3 and BT474 cells treated for 24 h with 10 µM or 1 µM of 1.
