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. 2009 Jan 28;29(4):1202–1211. doi: 10.1523/JNEUROSCI.4516-08.2009

Figure 5.

Figure 5.

Increase in proliferation in the SVZ during recovery requires Fgfr1. A–D, BrdU immunostaining in the SVZ at P17 of wild-type (A, B) or Fgfr1 cKO mice (C, D) under normoxia (A, C) or after hypoxia (B, D). Insets show high magnifications. E–H, Caspase-3 immunostaining in the SVZ at P10 of wild-type (E, F) or Fgfr1 cKO (G, H) mice under normoxia (E, G) or after hypoxia (F, H). I, J, Total number of BrdU+ cells (I) at P10 and P17 and caspase-3+ cells (J) at P10 in the SVZ by stereological analyses in wild-type (blue bars) and Fgfr1 cKO (red bars). Scale bar, 100 μm. N = 3 for each group. *p < 0.05 and **p < 0.01 by ANOVA with Sheffe post hoc test.