Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2009 Sep 15;101(8):1433–1443. doi: 10.1038/sj.bjc.6605316

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2009 Cancer Research UK

This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material.If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit https://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

FOXG1 inhibits p21^WAF1/CIP1 promoter activity on TGF-β treatment. (A) HEK293T, (B) A2780cp cells were transfected with fixed amount of the reporter plasmids pWWP and pRL-SV40 and varied amounts of pCMV2-Flag-FOXG1 and empty vector pCMV2. 100 pM TGF-β was added 24 h after transfection and was incubated for another 5 h before measuring the luciferase activity. The promoter activity of empty vector pCMV2 transfection was set to be 100% and the promoter activity with FOXG1 plasmids addition was expressed as a percentage relative to empty vector control.