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. 2009 Nov;134(5):363–367. doi: 10.1085/jgp.200910330

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© 2009 Striessnig

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jgp.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 1. — Mechanisms involved in lipid modulation of voltage-gated N- and L-type calcium channels by the slow pathway. The subunit structure of high threshold neuronal calcium channel complexes is illustrated together with a putative fatty acid regulatory domain (RD) that can also bind the palmitic acid side chains present in the N terminus of β2a subunits. For details see text. PLC, phospholipase C; PLA₂, phospholipase A₂; DAG-L, diacylglycerol lipase; PIP₂, phosphatidylinositol 4,5-bisphosphate; IP₃, inositol trisphosphate; β_np, nonpalmitoylated β subunit.