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. 2009 Oct 20;3:37. doi: 10.3389/neuro.08.037.2009

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Copyright © 2009 Wiltgen, Godsil, Peng, Saab, June, Van Linn, Cook, Houser, O'Dell, Homanics and Fanselow.

This is an open-access article subject to an exclusive license agreement between the authors and the Frontiers Research Foundation, which permits unrestricted use, distribution, and reproduction in any medium, provided the original authors and source are credited.

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(A) Schematic describing the electrode placement for experiments involving the thalamo-LA pathway. (B) The amount of lateral amygdala activity-dependent plasticity induced by 100 Hz stimulation of the thalamo-LA pathway in slices from floxed^+/+ controls (black circles) and α1^−/− (white circles) mice. Inset shows sample extracellular traces elicited during baseline (smaller response) and 25–30 min after 100 Hz stimulation in slices from floxed^+/+ controls (left side of panel) and α1^−/− mice (right side of panel). Calibration bars: 1 ms, 0.1 mV. (C) Schematic describing the electrode placement for experiments involving the LA-BLA pathway. (D) The amount of basolateral amygdala activity-dependent plasticity induced by 100 Hz stimulation of the LA-BLA pathway in slices from floxed^+/+ (black circles) and α1^−/− (white circles) mice. Inset shows sample extracellular traces elicited during baseline (smaller response) and 25–30 min after 100 Hz stimulation in slices from floxed^+/+ (left side of panel) and α1^−/− mice (right side of panel). Calibration bars: 1 ms, 0.1 mV.