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. Author manuscript; available in PMC: 2010 Feb 1.
Published in final edited form as: Arch Neurol. 2009 Feb;66(2):238–243. doi: 10.1001/archneurol.2008.585

Figure 3.

Figure 3

Five kindreds of 13 with H-BPN and identified with a conserved set of alleles (black bar) at 17q25 when analyzed by polymorphic markers [72GT1, 72GT2, 17S937, 17S939, GT1]. Haplotypes were generated assuming least numbers of crossovers, and prior knowledge of a disease associated set of alleles between affected families with H-BPN [9].