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. 2009 May 11;19(12):2827–2837. doi: 10.1093/cercor/bhp065

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© 2009 The Authors

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/2.0/uk/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 4. — LC stimulation–induced CA1 LTD is NR2B and β-adrenoceptor dependent. (A) NMDA receptor subunit 2B (NR2B)–selective antagonist ifenprodil (4.9 μg) and (B) β-adrenoceptor antagonist propranolol (2 μg) block LC stimulation–induced SC–CA1 LTD. (C, D, insets), Analog traces representing SC–CA1 field potentials before LC stimulation, 4 h and 24 h after LC stimulation. Top row: LC stimulation control. Bottom row: LC stimulation with drug application. Calibration: vertical bar, 1 mV; horizontal bar, 3 ms. The mean fEPSP slope is displayed along with the corresponding SEM.