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. 2009 Nov 24;4(11):e7989. doi: 10.1371/journal.pone.0007989

Table 1. Summary of clinical parameters of AML patients used in this study.

Patient ID FAB % CD34 Karyotype FLT3-ITD Responses to cytokines
2003-119 M0 80 N + c.a. TPO, IL-3, GM-CSF
2005-258 M0 73 -5q; +6 ND G-CSF, IL-3
2006-014 M1 19 N + TPO, IL-3 GM-CSF
2005-016 M1 65 N c.a., G-CSF
2004-168 M2 21 del (9) (q12;q22) + c.a., TPO, IL-3
2007-046 M2 20 N + TPO, G-CSF, IL-3
2003-152 M4 72 Inv (16) TPO, G-CSF, IL-3
2005-289 M5 95 N IL-3, GM-CSF, G-CSF
2003-114 M5 27 t(11;20) + c.a., G-CSF, GM-CSF
2007-272 M7 36 Complex c.a. TPO, IL-3

AML cells derived from peripheral blood (PB); Percentage of CD34+ (% CD34) in the total AML mononuclear cell fraction; FAB, French-American-British classifications; FLT3-ITD (internal tandem duplication) was present (+) or absent (−) in the AML cells; ND, not determined; N, normal; c.a. constitutive activation of STAT5.