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. 2009 Nov 21;15(43):5465–5471. doi: 10.3748/wjg.15.5465

Table 2.

Serum status of H pylori and CagA in cancer patients and control n (%)

Cancer
Control
Oral SCC Esophageal SCC Gastric cardia adenocarcinoma Gastric non-cardia adenocarcinoma Colon adenocarcinoma 1st hospital-based control 2nd hospital-based control Community-based control
n 199 317 29 167 240 305 403 395
H pylori
- 104 (52.3) 205 (64.7) 8 (27.6) 48 (28.7) 105 (43.8) 141 (46.2) 164 (40.7) 235 (59.5)
+ 95 (47.7) 112 (35.3) 21 (72.4) 119 (71.3) 135 (56.3) 164 (53.8) 239 (59.3) 160 (40.5)
CagA1
- 14 (14.7) 8 (7.3) 1 (4.8) 9 (7.6) 12 (8.9) 24 (14.6) - 11 (6.9)
± 13 (13.7) 10 (9.2) 3 (14.3) 8 (6.8) 7 (5.2) 14 (8.5) - 6 (3.8)
+ 68 (71.6) 91 (83.5) 17 (81.0) 101 (85.6) 116 (85.9) 126 (76.8) - 142 (89.0)
1

CagA status was determined among the subjects with H pylori seropositivity (No CagA data was available in 3 esophageal SCC, 1 gastric non-cardia adenocarcinoma and 1 community-based control).