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. 2009 Nov 3;3:9–20. doi: 10.2147/ppa.s2670

Table 1.

Drugs that affect sleep: Mechanisms and clinical implications

Drug class CNS Examples of drugs Affect on sleep architecture Potential mechanism Clinical implications
AEDs Phenobarbital, carbamazepine, phenytoin Very sedating. AEDs tend to ↑ TST, ↓ sleep latency. Inhibits neuronal calcium influx, adenosine receptor or 5HT activity. Sedation is dose-dependent, usually early on.
TCAs Amoxapine, amitriptyline, imipramine, nortriptyline, desipramine, doxepin, clomipramine Very sedating. Suppresses REM sleep, ↑ TST, ↑ stage 2 sleep. Antimuscarinic activity, α1-receptor stimulation. Suppressed REM sleep → ↓ motor inhibition → restlessness, psycho-motor agitation during sleep → subjectively ↓ sleep quality, ↑ daytime sleepiness.
Anxiolytic BzRAs* Alprazolam, lorazepam, chlordiazepoxide, diazepam, oxazepam. Very sedating. ↑ TST, ↓ sleep latency, ↓ SWS duration, ↓ REM, ↑ stage 2 sleep. GABA type A receptor stimulation. Minimize its use in daytime. Chronic BzRAs → ↓ SWS → long-term sequelae unknown.
SSRIs Sedating: paroxetine, fluvoxamine. Activating: fluoxetine, sertraline, citalopram. In general, SSRIs tend to ↑ TST, less sedating than TCAs. May ↓ REM, ↑ TWT, ↓ SE. 5HT activity. AM (if awakening) or PM (if sedating) dosing.
SNRI Venlafaxine Activating in some patients; sedating in 12–31%. ↓ TST. 5HT and NE activity. AM (if awakening) or PM (if sedating) dosing.
Lithium Lithium ↑ TST, ↑ SWS, ↑ stage 2 sleep, ↓ REM, ↓ REM latency. Dose at night.
Stimulants Ephedrine, pseudoephedrine, modafinil. Activating. ↓ TST, ↓ SWS, ↑ sleep latency. DOPA, NE, and 5HT activity. Avoid after 6 PM.
Anti-Parkinson Bromocriptine, levodopa. Sedating. Nightmares, ↓ SWS. DOPA Dose at night, if possible.
Cardiovascular
Lipophillic β-Blockers Propranolol, pindolol, metoprolol, timolol. Activating. ↑ awakenings, ↑ TWT, ↓ REM, nightmares. CNS β-blockade Daytime sedation if dosed in AM whereas hydrophillic agents (atenolol, sotalol) do not.
CNS Agents Norepinephrine, epinephrine Activating. ↓ REM, ↓ SWS. α1-receptor stimulation. Minimize its use, if possible, especially at night.
Dopamine Activating. ↓ REM, ↓ SWS. D2-receptor and α1-receptor stimulation. Minimize its use, if possible, especially at night.
α2-receptor agonist Clonidine ↑ stage 1, ↓ REM, nightmares. α2-receptor stimulation. α2-agonists → ↑ daytime sleep and sleepiness directly. May be helpful to dose at night.
α1-receptor blockers Doxazosin, prazosin, terazosin. α1-receptor inhibition. α1-receptor blockers → ↑ daytime sleepiness.
Ca++Channel Blockers Amlodipine, verapamil, nifedipine Exacerbate medical condition. ↓ Lower esophageal sphincter tone
→ nocturnal gastroesophageal reflux
→ sleep disturbance.
Diuretics HCTZ, furosemide. PM diuresis → frequent awakenings → ↓ sleep.
Analgesics
Opioids Codeine, morphine, hydrocodone. Sedating. ↓ SWS, ↓ REM. μ-receptor stimulation. Minimize its use, if possible, especially at night.
NSAIDs Ibuprofen, indomethcin, celecoxib. ↓ TST, ↓ SE. prostaglandin synthesis inhibition. Minimize its use, if possible, especially at night.
Other
Methylxanthine Theophylline Activating. ↑ stage 1, ↓ REM. Causes less restful sleep.
Antihistamines Diphenhydramine, promethazine. Sedating H1 receptor blockade. Minimize its use, if possible, especially at night.
Corticosteroids Dexamethasone, prednisone Activating. ↓ REM, ↓ SWS, nightmares. ↓ melatonin secretion. Can disrupt sleep, ↑ anxiety, induce mania or psychosis.
H2 Blockers Cimetidine, ranitidine, famotidine. Sedating. ↑ TST. H2 receptor blockade. Sedating if >60 years old, renal impairment.
Quinolones Ciprofloxacin, sparfloxacin, ofloxacin, grepafloxacin, levofloxacin Activating. GABA type A receptor stimulation. Linezolid rarely causes sleep disturbances.

Notes: Copyright @ 2006. Elsevier Ltd. Reproduced with permission based on Table 2 in Pandhardipande P, Ely EW. Sedative and analgesic medications: Risk factors for delirium and sleep disturbances in the critically ill. Crit Care Clin. 2006;22:313–327.

Abbreviations: AED, antiepileptic drugs; BzRAs, benzodiazepines; CNS, central nervous system; 5HT, serotonin, serotonergic; NE, norepinephrine; DOPA, dopamine; GABA, gamma-aminobutyric acid; H2, Histamine2-receptor; REM, rapid eye movement; MAOIs, monoamine oxidase inhibitors; NSAIDs, nonsteroidal anti-inflammatory drugs; SE, sleep efficiency; SNRI, serotonin norepinephrine reuptake inhibitor; SSRIs, selective serotonin reuptake inhibitors; HCTZ, hydrochlorothiazide; SWS, slow wave sleep (stage 3 and 4, or deep sleep); TCA, tricyclic and tetracyclic antidepressants; TST, total sleep time; TWT, total wake time; →, leads to or causes; ι, decrease or reduce; η, increase.