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. 2009 Dec 4;5(12):e1000755. doi: 10.1371/journal.pgen.1000755

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Shor et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Unlike HMR-E, orc2-1-resistant replication origin ARS1005 could fire with high efficiency at its endogenous chromosomal location. Like HMR-E, ARS1005 was resistant to orc2-1 for firing efficiency, while neighboring origin ARS1006 that was orc2-1-sensitive for ORC binding was also orc2-1–sensitive for firing.