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. 2009 Nov 2;187(3):429–442. doi: 10.1083/jcb.200904049

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© 2009 Brunelle et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jcb.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 1. — MCL-1 overexpression targeted to hematopoietic cells by the H2K promoter in conjunction with c-myc results in the development of leukemia in mice. (A) Levels of MCL-1 protein expression in wild-type and H2K-Mcl-1 mouse spleenocytes. (B) Kaplan-Meier plot showing the survival in number of days for Mcl-1/Eμ-Myc, H2K-Mcl-1 only, Eμ-Myc only, and wild-type mice. This is from a cohort of mice containing seven of each genotype. (C) Number of white blood cells per μl of blood from Mcl-1/Eμ-Myc, H2K-Mcl-1 only, Eμ-Myc only, and wild-type mice. This is from a cohort of mice containing seven of each genotype.