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. 2008 Aug;4(3):157–169. doi: 10.2174/157340308785160606

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©2008 Bentham Science Publishers Ltd.

This is an open access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.5/) which permits unrestrictive use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. (4) — Colocalisation of BNP and Chromogranin A during CHF (A), which is considerably decreased by unloading (B). Normal control hearts are devoid of this colocalisation (C). Red arrow indicates Chromogranin A, green arrow indicates BNP and yellow arrows indicate colocalisation of Chromogranin A with BNP.

Despite being significantly decreased in the myocardium after unloading, owing to its low expression in the myocardium, Chromogranin A plasma levels do not reflect hypertrophy regression after LVAD (D). ANP and BNP are significantly decreased in the plasma after unloading (E and F).