Table 2. Clinical outcomes and the presence of different genes in cagI and cagII.

	Gastritis (n = 182)	Mild gastritis (n = 34)	PUD (n = 41)	GC (n = 8)	Total (n = 231)
cagA+	127 (69.7%)	26 (76.5%)	22 (53.6%)	3 (62.5%)	154 (66.7%)
cagE+	71 (39.0%)	13 (38.2%)	18 (43.9%)	1 (12.5%)	90 (39.0%)
cagT+	53 (29.1%)	12 (35.3%)	15 (36.5%)	2 (25.0%)	70 (30.3%)
cagA+/cagE+/cagT+ (1)	32 (17.5%)	9 (28.1%)	8 (19.5%)	0 (0%)	40 (17.3%)
cagA+/cagE-/cagT+ (2)	12 (6.5%)	2 (5.9%)	2 (4.8%)	1 (12.5%)	15 (6.4%)
cagA-/cagE+/cagT+ (2)	3 (1.6%)	0 (0.0%)	1 (2.4%)	1 (12.5%)	5 (2.1%)
cagA-/cagE-/cagT+ (2)	6 (3.3%)	1 (2.9%)	4 (9.8%)	0 (0.0%)	10 (4.3%)
cagA+/cagE+/cagT- (3)	24 (13.2%)	3 (8.8%)	6 (14.6%)	0 (0.0%)	30 (13.0%)
cagA-/cagE+/cagT- (3)	12 (6.6%)	1 (2.9%)	3 (7.3%)	0 (0.0%)	15 (6.5%)
cagA+/cagE-/cagT- (3)	59 (32.4%)	12 (35.3%)	6 (14.6%)	4 (50%)	69 (29.9%)
cagA-/cagE-/cagT- (4)	34 (18.7%)	6 (17.6%)	11 (26.8%)	2 (25%)	47 (20.3%)

Mild gastritis was defined as gastritis with no acute inflammation, none to mild chronic inflammation as well as no gastric atrophy/intestinal metaplasia.

cagA+: cagA-positive, cagA-: cagA-negative, cagE+: cagE-positive, cagE-: cagE-negative, cagT+: cagT-positive, cagT-: cagT-negative

(1) cagA+/cagE+/cagT+ = triple-positive

(2) cagI+/cagII+, but one of cagI gene was negative

(3) cagI+/cagII-

(4) all negative (cagI-/cagII-) = triple-negative