In an analysis of occipital cortex (BA 17,18), parietal cortex (BA 41,42); amygdala (top, bottom), BPD-IED patients showed significantly more relative glucose metabolism (rGMR) in amygdala than controls, while controls showed significantly higher rGMR in the occipital cortex during the provoked compared with the non-provoked condition (*post-hoc tests, p<.05).