Figure 1.
Representative recordings from one myocardial infarction (MI) rat (fractional shortening = 17%) of tension developed within the left triceps surae muscles, arterial pressure (AP), heart rate (HR) (bpm = beats min−1), renal sympathetic nerve activity (RSNA), and the relative changes in RSNA during 30 s continuous contraction of the left hindlimb muscles before (left) and after (right) intra-arterial injection of tempol (10 mg) into the hindlimb circulation. Note that RSNA increased rapidly towards peak values from the onset of muscle tension development, and that the elevated RSNA gradually decreased towards baseline level.