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. 2009 Dec 29;4(12):e8485. doi: 10.1371/journal.pone.0008485

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This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration, which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

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(A) CX₃CR1⁺ and CX₃CR1⁻ GC B cells were analyzed after staining with mAbs. Results are percent positive cells, minimum to maximum ranges from ten independent experiments. (B) V_H5(D)J_H-μ rearrangement sequences were evaluated in CX₃CR1⁺ and CX₃CR1⁻ GC B cells. 104 molecular clones from five different CX₃CR1⁺ GC B cell fractions were compared to 63 molecular clones from three different CX₃CR1⁻ GC B lymphocyte fractions. 34 molecular clones from three different CD10^-CD27⁻ naive B cell fractions were tested as controls. Results are ratios between number of mutations and number of molecular clones.