. Author manuscript; available in PMC: 2010 Dec 1.

Published in final edited form as: J Acquir Immune Defic Syndr. 2009 Dec 1;52(4):480–487. doi: 10.1097/qai.0b013e3181b939e5

Table 3.

Multivariate analysis of the association of lipid parameters and hsCRP, including BMI in the model.

	Regression coefficient (95% CI)^a	p-value^b
Total cholesterol (unit=50)	1.38 (1.03-1.90)	0.035
LDL (unit=50)	1.60 (1.05-2.43)	0.029
Non-HDL (unit=50)	1.47 (1.07-2.02)	0.018
HDL (unit=10)	0.81 (0.66-1.00)	0.050
Triglycerides (unit=200)	1.31 (1.05-1.60)	0.019

Regression coefficients were obtained by taking the exponential of β-coefficients from the model, indicating a relative increase (percent) in hsCRP values by one unit increase of the factors listed in the table.

Effect of HDL cholesterol was adjusted for non-HDL cholesterol and the variables listed in Table 1 (age, PI use, CD4⁺ T cells, HIV-1 RNA, race, sex, smoking status, fasting status, BMI, and use of lipid-lowering therapy) and time. LDL was adjusted for HDL cholesterol and non-HDL cholesterol was adjusted for HDL cholesterol along with the set of covariates in Table 1. Cholesterol and triglycerides were adjusted for only the set of covariates listed in Table 1.

Note: CI = confidence interval, hsCRP = highly sensitive c-reactive protein, BMI = body mass index, LDL = low-density lipoprotein, HDL = high-density lipoprotein