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. 2009 Nov 2;78(1):88–99. doi: 10.1128/IAI.00985-09

FIG. 2.

FIG. 2.

B. mallei tssE mutants exhibit apparent growth defects in RAW 264.7 macrophages. Monolayers were infected with the various B. mallei strains at an MOI of 1. Bacterial uptake (white bars) and intracellular survival (black bars) were quantitated at 3 h and 24 h postinfection, respectively. (A) Uptake and survival phenotypes of B. mallei parent and mutant strains. (B) Complementation analysis of B. mallei BM0739 harboring pA0739 (tssE+) or pBHR1 (vector control). Values represent the means ± standard deviations (SDs) of three independent experiments.