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. 2009 Jun;26(6):861–876. doi: 10.1089/neu.2008.0645

Table 2.

Summary of Injury Mechanisms from Secondary bTBI

Reference In-vitro injury RM IM IC O Summary
Regan and Choi, 1994 Stylus X X     Injury mediated by excessive NMDAR  activation and dihydropyridine-sensitive  mechanisms
Regan and Panter, 1995 Stylus X     X Injury was reduced with MK801 and  free radical scavengers
Mukhin et al., 1997a, 1998 Stylus X       Glutamate receptor antagonists were  neuroprotective
Mukhin et al., 1997b;  Mukhin et al., 1996 Stylus X       Activation of mGluR1 exacerbates injury
Faden et al., 1997 Punch laceration (stylus) X       Group III mGluR agonists are neuroprotective
Wang et al., 2005 Injured tissue from  humans/rodents       X Performed proteomics on injured tissue to  determine biomarkers of injury
Lucas et al., 1985;  Emery et al., 1987;  Kirkpatrick et al., 1985 Laser transection       X Damage to cytoskeleton, neurofilaments  microtubules, organelles (especially  mitochondria)
Lucas et al., 1991 Laser transection   X     At low [Ca2+], Na+ mediates injury
Rosenberg et al., 1996a Laser transection       X MPSS helps stabilized damaged membranes
Rosenberg et al., 1996b;  Rosenberg et al., 2001 Laser transection   X     Na+ and Cl ions may mediate injury
Balentine et al., 1988 Weight drop       X In vitro injury is similar to in vivo spinal  cord trauma
Epstein, 1971 Spinner bottle       X Determined viability and oxidative metabolism  of rat cortices after stir-bar-induced trauma

RM, receptor mediated; IM, ion mediated; IC, intracellular signaling; O, other.