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. Author manuscript; available in PMC: 2010 Jan 12.
Published in final edited form as: Eur J Neurosci. 2009 Jan;29(2):287–306. doi: 10.1111/j.1460-9568.2008.06590.x

Fig. 4. D2-like DA receptors, β-adrenergic receptors and NMDA receptors are not required in DA and SKF R-38393-induced ERK activation in striatal neurons.

Fig. 4

Striatal neurons were pretreated for 15 min with vehicle or with the following compounds: spiperone (10 μM), propranolol (10 μM) or MK801 (10 μM), and then treated with 10 μM of either DA [A] or SKF R-38393 [B] for 20 min. Immunoblot analysis of p-ERK1/2 expression was conducted; data shown are the average ± SEM of three independent experiments (n=3). In bar graph, P<0.05 (*), represents the result of paired Student’s t test with 4 degrees of freedom for the corresponding agonist (DA or SKF R-38393) alone-treated group; p<0.05 (#), represents the result of paired Student’s t test with 4 degrees of freedom for the corresponding control group.