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. 2009 Dec 21;206(13):2869–2877. doi: 10.1084/jem.20090845

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© 2009 Conrad et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jem.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 1. — Prenatal A. lwoffii F78 exposure significantly prevented the extent of the asthmatic phenotype in the offspring. White bars represent offspring exposed prenatally to control PBS, and black bars represent offspring exposed prenatally to A. lwoffii F78. (A) Differential leukocyte numbers in the BAL of offspring. (B) Representative lung tissue cross sections stained with PAS to visualize mucus-producing goblet cells (arrows) and airway inflammation in offspring. Bars, 100 µm. (C) Quantification of mucus-producing goblet cells, mucus volume, and inflammation in offspring airways. (D) Offspring airway responsiveness to MCh. Results represent one out of two independently performed experiments with similar outcomes (n ≥ 8 per group). Means ± SEM are shown. *, P < 0.05; **, P < 0.01; ***, P < 0.001.