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. 1998 Dec 22;95(26):15700–15705. doi: 10.1073/pnas.95.26.15700

Figure 4.

Figure 4

The V domain of HIgR is sufficient to mediate HSV-1 entry into J1.1–2 cells. J1.1–2 cells were transfected with a deleted form of PRR1 consisting of the V domain fused to its transmembrane and cytoplasmic regions, designated as V-TM(PRR1) (A and B), or with a chimeric receptor consisting of the V domain of HIgR/PRR1 fused to the CC-transmembrane–cytoplasmic regions of hPVRα, designated as V(HIgR)-PVRα (C and D), and for comparison with HIgR (E). (B and D) Blocking effect of mAb R1.302 on R8102 infectivity.