Skip to main content
. Author manuscript; available in PMC: 2011 Feb 5.
Published in final edited form as: Circ Res. 2009 Nov 19;106(2):346. doi: 10.1161/CIRCRESAHA.109.205724

Figure 6. Absence of dystrophin delays the onset of ventricular dysfunction in Lap2α−/−/Mdx mice.

Figure 6

FS% values in young male Lap2α−/−/Mdx mice are higher compared to Lap2α−/− animals, whereas old mice of both genotypes show similar systolic dysfunction (ANOVA; p<0.05 for (a) WT vs. KO, (b) WT vs. Mdx and (c) WT vs. Lap2α−/−/Mdx mice; (n) - sample size; mean ± SE).