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. 2010 Feb 8;188(3):369–381. doi: 10.1083/jcb.200905057

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© 2010 Thompson and Compton

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jcb.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 6. — Inhibition of p38 stress kinase but not MAPK allows proliferation of aneuploid cells. (A and B) HCT116 cells with wild-type p53 and RPE1 cells were treated as described in Fig. 5. After shake-off, cells were grown in the presence of p38 inhibitor (SB203580; A) or MAPK inhibitor (PD98059; B) for the remainder of the experiment. For each time point/condition, 600 nuclei were counted per chromosome. Bars represent the mean percent deviation from the modal chromosome number, and error bars show SEM. *, P < 0.05; χ² test.