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Wound closure, facilitated by unmodified CD133+ CCM, is retarded as compared to vehicle, when Wnt antagonist sFRP-1 is added (A). Treatment of wounds with sFRP-1 in the absence of CD133+ CCM did not modulate wound closure (A). Likewise, capillarization of wounds was supported by CD133+ CCM only in the absence of sFRP-1 (B) (n=7 per group). *P<0.05, **P<0.01, ***P<0.005 vs NCCM, +++P<0.005 vs CD133+ CCM.
