Fig. 7. Proposed mechanism on how β-tubulin plays a role in the AhR signaling.
By interacting with Arnt in the cytoplasm, β-tubulin controls the Arnt content in the nucleus. Increased cytoplasmic β-tubulin content reduces the nuclear Arnt content, causing a reduction of the amount of the AhR/Arnt heterodimer formation upon ligand activation. Activation of the AhR-dependent gene transcription is compromised when the cytoplasmic β4-tubulin content increases.