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Activation of NF-κB by TLR2 mutants in response to a synthetic di-acylated lipopeptide MALP-2 and BCGLM. Mutants defective for responses to Pam3CSK4-sCD14 were tested for responses to MALP-2-sCD14 complexes (A) and BCGLM-sCD14 complexes (B) as described for Fig. 2. On each panel, responses mediated by the indicated mutant (○) are compared with those of wild type TLR2 (●).
