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. Author manuscript; available in PMC: 2010 Feb 24.
Published in final edited form as: Differentiation. 2008 Oct 16;77(1):103–111. doi: 10.1016/j.diff.2008.09.010

Fig. 2.

Fig. 2

(A) Trp53−/− tissues recombinant showing normal prostate gland with moderate nuclear atypia (arrows) and severe nuclear atypia (inset). (B) PIN III in Pten−/− tissue recombinant showing large vacuolated cells with abundant cytoplasm characteristic of Pten disruption. (C) PIN IV in third-generation Pten/Trp53 double heterozygous tissues showing severe nuclear atypia characteristic of Trp53 disruption.