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. Author manuscript; available in PMC: 2010 Mar 3.
Published in final edited form as: J Neuropathol Exp Neurol. 2008 Jul;67(7):677–686. doi: 10.1097/NEN.0b013e31817e5c5e

Table 1.

TABLE 1A. Summary of Cerebral White Matter Pathology in PVL cases
Case* GA PNA PCA Focal Acute Necrosis Focal Organizing Necrosis Focal Scar w/Mineral Diffuse Gliosis ↓WM and/or ↑Vents Hypo-myelin
1^ 0054 35** 0 35 + + + + +
5^ 9841 34** 0 34 + + +
6^ 9636 28** 7 35 + + + + +
7^ 9829 36** 0 36 + + +
8 0006 39 1 40 + +
9 9615 35** 5 40 + +
10 9520 36** 6 42 + + + +
11 0053 40 4 44 + +
12 0109 28** 20 48 + + + +
13^ 9817 36** 15 51 + + + + +
14 9627 31** 64 95 + + +
TABLE 1B. Continued Summary of the Neuropathology of PVL Cases
Case* Bst CB BG Thal Hipp CCH WMH SAH GMH Diagnosis
1^ A, B A Potter syndrome; abnormal temporal lobe gyration; malrotation of hippocampi; duplication of dentate gyrus
5^ G G + Osteogenesis imperfecta
6^ A E,H E,H + Necrotizing enterocolitis; bowel perforation; coagulopathy; sepsis
7^ A I + CMV infection in utero; ABO incompatibility; dwarfism/skeletal dysplasia; fetal heart failure
8 A B Urea cycle defect (ornithine transcarbamylase deficiency); hyperammonemia
9 NA J A A A K + Breech birth; oligohydramnios; renal sclerosis
10 A L A A A Intrauterine growth retardation; breech birth; oligohydramnios; dysplastic kidneys; malformed basal ganglia and hippocampus
11 A A NA A, M A Congenital cardiac malformation; ECMO treatment
12 A Pulmonary hypertension; pneumonia; ECMO treatment
13^ N Breech birth; oligohydramnios; pneumothorax; respiratory failure
14 A + + Congenital cardiac malformation; ECMO treatment
*

Case numbers are the same as those used in the prior analysis of white matter (15). Cases 2, 3, and 4 from that study were not included here;

^

Case was included in a previous gray matter survey (3); GA = gestational age in weeks; PNA = postnatal age in weeks; PCA = postconceptional age in weeks; mineral = mineralization; WM = white matter, Vents = ventricles.

**

Prematurity is defined as a gestational age less than 37 weeks; +, present; −, absent.

*

Case numbers are the same as those used in the prior analysis of white matter (15) (see Materials and Methods);

^

Case was included in prior gray matter survey (3); +, pathology present; −, pathology absent; A = reactive gliosis; B = neuronal loss; C = ischemic necrosis; D = hemorrhagic infarct; E = apoptosis; F = focal microhemorrhage; G = mineralization; H = perivascular hemorrhage; I = microglial nodules; J = vermis cortical scar; K = organizing infarct; L = Purkinje cell loss; M = microinfarct; N = necrotic focus in hypothalamus; ECMO = extracorporeal membrane oxygenation; NA = not available; Bst, brainstem; CB, cerebellum; BG, basal ganglia; Thal, thalamus; Hipp, hippocampus; CCH, cerebral cortex hemorrhage; WMH, white matter hemorrhage; GMH, germinal matrix hemorrhage.