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. 2010 Feb;21(2):327–336. doi: 10.1681/ASN.2009010072

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Copyright © 2010 by the American Society of Nephrology

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Figure 1. — Histopathology after renal IRI is shown. (A) Acute tubular damage, shown as percentage of damage to total tubular area, was quantified 1, 3, 7, and 30 d after 30 min of IRI of the left kidney in Terc^+/+, G1 Terc^−/−, and G4 Terc^−/− kidneys. (B) Representative periodic acid-Schiff stainings 3 d after IRI showing more acute tubular damage in G4 Terc^−/− compared with G1 Terc^−/− and Terc^+/+. * areas of acute tubular damage. (C) Chronic tubular deterioration and (D) interstitial fibrosis—both reflecting chronic kidney damage—were quantified 1, 3, 7, and 30 d after IRI in Terc^+/+, G1 Terc^−/−, and G4 Terc^−/−. (E) Representative Masson Trichrome stainings showing increased interstitial fibrosis (blue staining, indicated by black arrowhead) in G4 Terc^−/− on day 30 after IRI. □, Terc^+/+; , G1 Terc^−/−; ■, G4 Terc^−/−. Data are means ± SEM; significances are indicated. Magnification, ×200.

Inline graphic — Histopathology after renal IRI is shown. (A) Acute tubular damage, shown as percentage of damage to total tubular area, was quantified 1, 3, 7, and 30 d after 30 min of IRI of the left kidney in Terc^+/+, G1 Terc^−/−, and G4 Terc^−/− kidneys. (B) Representative periodic acid-Schiff stainings 3 d after IRI showing more acute tubular damage in G4 Terc^−/− compared with G1 Terc^−/− and Terc^+/+. * areas of acute tubular damage. (C) Chronic tubular deterioration and (D) interstitial fibrosis—both reflecting chronic kidney damage—were quantified 1, 3, 7, and 30 d after IRI in Terc^+/+, G1 Terc^−/−, and G4 Terc^−/−. (E) Representative Masson Trichrome stainings showing increased interstitial fibrosis (blue staining, indicated by black arrowhead) in G4 Terc^−/− on day 30 after IRI. □, Terc^+/+; , G1 Terc^−/−; ■, G4 Terc^−/−. Data are means ± SEM; significances are indicated. Magnification, ×200.