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. 2010 Jan 27;9:5. doi: 10.1186/1475-2840-9-5

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Copyright ©2010 Vahtola et al; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Increased p38-phosphorylation in diabetic GK rats 12 weeks after MI. Panel (A) shows an increase in total myocardial p38 protein expression in post-MI GK and Wistar rats. Panel (B) shows an increase in phosphorylated p38 in GK rats with MI. The lower panels in (A) and (B) shows representative western blots of total--p38 and phospho-p38 respectively, anti-tubulin was used as loading control. Panel (C) shows the mRNA expression of connective tissue growth factor (CTGF) in myocardial tissue and (D) shows the mRNA expression of interleukin -6 (IL-6) in myocardial tissue. Data is presented as means ± SEM; * indicates P < 0.05 vs. W (sham-operated Wistar), ^#indicates P < 0.05 vs. GK (sham-operated GK), ^†indicates P < 0.05 vs. W+MI.