Skip to main content
. 2010 Feb 11;51(3):354–365. doi: 10.1093/pcp/pcq016

Fig. 3.

Fig. 3

Complementation of the ABA-insensitive phenotype of atalmt12-1 with genomic AtALMT12 sequences. Plants were treated with or without 1 μM ABA and stomatal closure was assayed. Complementation of atalmt12-1 by the native promoter (NP)-driven genomic sequence of AtALMT12 with or without GFP fusion (NP::genome, NP::genome–GFP; n = 5–6 independent experiments), and the NP-driven coding sequence (NP::ORF, n = 8 independent experiments).

HHS Vulnerability Disclosure