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. Author manuscript; available in PMC: 2010 Mar 12.
Published in final edited form as: Integr Cancer Ther. 2009 Nov 18;8(4):321–328. doi: 10.1177/1534735409352027

Figure 6. Expression of genes related to circadian circuitry in cigarette smoke (CS)-exposed lungs of C57BL6 mice.

Figure 6

Three-month-old male mice were exposed to whole smoke (WS), 6 hours a day for a period of 10 days at 90 mg/m3 of total suspended particles (TSP), and then increased to 125 mg/m3 over 5 days, and continued for 3 additional days at 125 mg/m3. For filtered smoke (FS) exposure, WS was drawn through a high-efficiency particulate air (HEPA) filter generating FS before entering the exposure chamber. *P < .05 and **P < .01 represent level of significance in FS-exposed group as compared with the respective air-group. #P < .05, ##P < .01, and ###P < .001 represent level of significance in WS-exposed group as compared with respective air-breathing controls. Data are represented as mean ± standard error of the mean, n = 5 to 6.