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. 2010 Jan 8;298(3):H1088–H1095. doi: 10.1152/ajpheart.01280.2008

Fig. 2.

Fig. 2.

Right: reduced cardiomyocyte apoptosis (A) and increased myocyte cross-sectional area (B) in mPer2-M mouse hearts 4 days after chronic infarction are shown. Left: there were 15% less terminal deoxynucleotide transferase-mediated dUTP nick end labeling (TUNEL)-positive apoptotic cardiomyocytes observed in mPer2-M mice (top, right) compared with WT (top, left; *P < 0.05). The average myocyte cross-sectional area (in μm2; †P < 0.001) was increased in mPer2-M mice (bottom, right) vs. WT (bottom, left).