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. Author manuscript; available in PMC: 2011 Apr 1.
Published in final edited form as: Thromb Res. 2010 Feb 13;125S1:S7–S10. doi: 10.1016/j.thromres.2010.01.022

Fig. 1.

Fig. 1

Residues involved in experimental contacts are mapped onto the Se model. TF: (Lys165, Lys166) [20–25], (Tyr157, Lys159, Ser163, Gly164) [23,25], Tyr185 [24,25], (Asn199, Asp204) [24], Arg200 [9,24,26,27], Lys201 [9,27], (Leu104, Thr197) [27]; FVIIa: Arg 36 [28], SP((Val21, Glu154, Met156)[29,30], Leu144 [30] Ala152 [30,31], Arg147 [32]); FXa [7]: Glu51, Asn57, Asp92, Asp95, SP(Lys134, Asp185A, Lys186).