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. Author manuscript; available in PMC: 2010 Nov 1.
Published in final edited form as: J Mol Cell Cardiol. 2009 Jul 23;47(5):723–729. doi: 10.1016/j.yjmcc.2009.07.014

Fig. 6.

Fig. 6

SLN is a target for CaMKII phosphorylation. (A) In vitro phosphorylation of mouse and human SLN by CaMKII — representative autoradiogram showing phosphorylated SLN corresponding to mouse and human SLN. (Details are in Section 2 Materials and methods). (B) Effect of CaMKII inhibitor autocamtide inhibitor peptide (AIP) on myocyte contractility — Contractility of control and Ad.SLN-WT infected myocytes was measured in response to isoproterenol in the presence and absence of 20 μM AIP. Data expressed as mean±S.E.M. * Indicates that contractility is significantly different from corresponding untreated myocytes. (p<0.05).