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. 2010 Mar 23;5(3):e9792. doi: 10.1371/journal.pone.0009792

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Han et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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(A) Percentage of B220⁺, MHC class II⁺, CD4⁺, CD8⁺, CD4 and CD8 double negative cell populations in splenocytes from gp96tm transgenic female mice treated with vehicle (n = 9), GPM1 (n = 9), and dexamethasone (n = 7) at 30mg/kg. (B) Percentages of B220⁺, MHC class II⁺, CD4⁺, CD8⁺, CD4 and CD8 double negative cell populations in lymph nodes from gp96tm transgenic female mice treated with vehicle (n = 9), GPM1 (n = 9), and dexamethasone (n = 7) at 30mg/kg. (C) Percentage of CD4⁺CD44^high (memory T cells), CD4⁺CD62L^low (effector T cells), and CD4⁺CD69⁺ (activated T cells) in splenocytes from gp96tm transgenic female mice treated with vehicle (n = 9), GPM1 (n = 9), and dexamethasone (n = 7) at 30mg/kg. (D) Percentages of CD4⁺CD44^high, CD4⁺CD62L^low, and CD4⁺CD69⁺ lymph node cells in vehicle- (n = 9), GPM1- (n = 9), and dexamethasone- (n = 7) treated mice. Student t-test P values comparing GPM1-treated mice with control are indicated.