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. 2010 Mar 3;65A(4):344–352. doi: 10.1093/gerona/glq018

Figure 7.

Figure 7.

Peroxisome proliferator–activated receptor (PPAR) γ in Ames dwarf (df) mice after growth hormone (GH)/thyroxine (T4) treatment. (A) Hepatic PPARγ messenger RNA (mRNA) in young-df saline, n = 12; old-df saline, n = 10; young-df GH and T4, n = 12; old-df GH and T4, n = 10; young-N, n = 13; and old-N n = 10. (B) Hepatic PGC-1α mRNA in young-df saline, n = 12; old-df saline, n = 10; young-df GH and T4, n = 12; old-df GH and T4, n = 7; young-N, n = 12; and old-N, n = 9. (C) Hepatic glucose transporter-2 mRNA in young-df saline, n = 12; old-df saline, n = 9; young-df GH and T4, n = 10; old-df GH and T4, n = 5; young-N, n = 12; and old-N, n = 8. Nondetectable levels in old-df GH and T4 resulted in low n values. Groups that do not share a superscript are significantly different (p < .05).