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. Author manuscript; available in PMC: 2010 Apr 12.
Published in final edited form as: Arch Virol. 2010 Jan 6;155(3):367–370. doi: 10.1007/s00705-009-0579-2

Table 5.

Mutations by case status

SNP* CIN2+
n = 16 (%)
Persistent
n = 19 (%)
Clearance/control
n = 17 (%)
HPV16
  A51G 0 0 1 (5.9)
  T171C 2 (12.5) 3 (15.8) 2 (11.8)
  T228C 3 (18.8) 3 (15.8) 2 (11.8)
  T234G 3 (18.8) 3 (15.8) 2 (11.8)

SNP n = 13 (%) n = 22 (%) n = 12 (%)

HPV31
  G21A 6 (46.2) 9 (40.9) 2 (16.7)
  C67T 1 (7.7) 3 (13.6) 4 (33.3)
  C86T 0 2 (9.1) 0
  C111T 3 (23.1) 15 (68.2) 6 (50.0)
  G136A 3 (23.1) 14 (63.6) 6 (50.0)
  A184G 13 (100.0) 22 (100.0) 12 (100.0)
  G186C 0 1 (4.5) 2 (9.1)

SNP n = 0 n = 4 (%) n = 14 (%)

HPV73
  A92G 2 (50.0) 10 (71.4)
  C168T 4 (100.0) 13 (92.9)
  G240T 4 (100.0) 13 (92.9)
  A253G 4 (100.0) 12 (85.7)
*

SNP single nucleotide polymorphism